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What is is LSD?
LSD is the most common hallucinogen and is one of the most potent mood-changing chemicals. It is manufactured from lysergic acid, which is found in ergot, a fungus that grows on rye
LSD is usually found on “blotter” paper (paper that is perforated into small squares). The squares or “tabs” may be colored or have images printed on them. Liquid LSD is a clear liquid, usually in a small container, tube or flask. LSD can also be found in thin squares of gelatin.
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P-LSD 100mcg Blotters is sold for research purposes only and is not be utilized for any other purposes, including, but not limited to, in vivo diagnostic purposes, in foods, in drugs, in medicinal devices, and/or cosmetics for humans and/or animals.
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2′-Oxo-PCE (Eticyclidone, O-PCE) is an example of a contemporary designer drug specifically chosen to mimic and/or replace the functional and structural features of its predecessors. 2′-Oxo-PCE (Eticyclidone, O-PCE) is being marketed and sold as a designer drug replacement for MXE and other dissociatives for recreational purposes.
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25B-NBOMe or 2C-B-NBOMe is a serotonergic N-benzyl derivative of the substituted phenethylamine psychedelic known as 2C-B. 25B-NBOMe is a substituted phenethylamine with methoxy groups CH3O- attached to carbons R2 and R5 as well as a bromine atom attached to carbon R4. It differs from 2C-B structurally through a substitution on the amine (NH2) with a 2-methoxybenzyl (BOMe) group. 25B-NBOMe shares this 2-methoxybenzyl substitution with other chemicals of the NBOMe family. This NBOMe addition contains a methoxy ether CH3O- bound to a benzene ring at R2
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25C-NBOMe had no history of human use before being sold online as a designer drug in 2010. Extremely little is known about the pharmacological properties, metabolism, and toxicity of 25C-NBOMe in humans, and it has been associated with many deaths and hospitalizations. Along with its highly sensitive dose-response and unpredictable effects, many reports also suggest that this substance may be overly difficult to use safely. Therefore it is highly advised to approach this poorly understood psychedelic substance with the proper amount of precaution and harm reduction practices if choosing to use it.
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Among psychedelics, this compound is considered to be pharmacologically unique in terms of the high potency, affinity, and selectivity with which it binds to the 5-HT2a receptor. Contrary to popular belief, it is not a “full agonist”, although questions have been raised about how the effects it produces differ from other 5-HT2a partial agonists, which include the range of traditional psychedelics.
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The general public has long been aware of traditional illicit drugs such as cocaine, marijuana, and methamphetamine. Designer drugs were created in the 1960s to avoid the provisions of existing drug laws by preparing analogs or derivatives of currently available drugs. Designer drugs including the 2C class discussed in this article are not new but have been increasing in popularity. Manufacturers of designer drugs purposely change the structure of these drugs to stay ahead of drug laws, making it difficult for clinicians to stay informed. Structure manipulation to produce new compounds, advertisement of these compounds as “legal” highs, and ease of access of designer drugs from the internet, raves, night clubs, and sale in head shops have rejuvenated and promoted the current designer drug resurgence [1, 2].
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Drugs in the 2C family usually come in the form of capsules or tablets that are swallowed or a powder form that is snorted. They may also come in liquid form.
Typical doses of 2C drugs usually range from 10 to 30 milligrams. The effects of 2C-E typically last for eight to 12 hours, but some 2C drugs can last for 24 hours or longer. Snorting the drugs causes a more rapid onset of effects than swallowing them does.
Occasionally, people mix 2C drugs with other hallucinogens. The combination of 2C-B and LSD is known as a banana split, while mixing 2C-B and MDMA is called a party pack
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In the body, 4-AcO-DMT / psilacetin is thought to be deacetylated into psilocin during first pass metabolism and subsequent passes through the liver (evident as psilacetin is also active when injected). This has not been formally proven, however, and is based on reports that most users cannot tell the difference between these two compounds when ingested to the point that they are often considered as indistinguishable from each other in terms of their subjective effects.